Mechanisms of Reciprocal Interactions between HIV Associated Neuroinflammation and CNS Persistence: Implications in HIV Neuropathogenesis and Cure (R21 Clinical Trial Not Allowed)
RFA-MH-25-181This grant opportunity aims to explore how interactions between HIV-related brain inflammation and the persistence of the virus in the central nervous system occur, even when virus levels are well-controlled. It will use advanced models and technologies, such as innovative brain cell systems, organoid models, and single-cell analysis methods, to conduct this research.
The goals of this initiative are to examine mechanisms of reciprocal interactions between HIV-associated neuroinflammation and CNS persistence in the setting of excellent virologic control using novel CNS cell systems, organoid models and single cell technologies
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